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Noonan Syndrome   (Spanish Version)  

Definition:

Noonan syndrome is a genetic disorder that causes abnormal development of multiple parts of the body. It used to be called Turner-like syndrome because certain symptoms (webbing of neck and abnormally shaped chest) resembled those seen in Turner syndrome.



Alternative Names:


Causes, incidence, and risk factors:

Defects in four genes (KRAS, PTPN11, RAF1, SOS1) can cause Noonan syndrome. About half of those affected by Noonan syndrome have a PTPN11 mutation. Persons with a defect in the KRAS gene have a more severe form of Noonan syndrome. Those with defects in the RAF1 gene tend to have a particular heart problem (hypertrophic cardiomyopathy). Problems with these genes cause certain proteins involved in growth and development to become overactive.

Noonan syndrome is inherited, which means it is passed down through families. It is an autosomal dominant condition. This means that only one parent has to provide the faulty gene for the baby to have the syndrome. However, the fact that some children do not have a parent with Noonan syndrome likely means that some cases are not inherited.



References:

Rapaport R. Hypofunction of the Ovaries. In: Kliegman RM, Behrman RE, Jenson HB, Stanton BF, eds. Nelson Textbook of Pediatrics. 18th Ed. Philadelphia, Pa: Saunders Elsevier; 2007: chap 587.




Review Date: 8/26/2009
Reviewed By: Chad Haldeman-Englert, MD, Wake Forest University School of Medicine, Department of Pediatrics, Section on Medical Genetics, Winston-Salem, NC. Review provided by VeriMed Healthcare Network. Also reviewed by David Zieve, MD, MHA, Medical Director, A.D.A.M., Inc.


The information provided herein should not be used during any medical emergency or for the diagnosis or treatment of any medical condition. A licensed physician should be consulted for diagnosis and treatment of any and all medical conditions. Call 911 for all medical emergencies. Links to other sites are provided for information only -- they do not constitute endorsements of those other sites. Copyright 2003 A.D.A.M., Inc. Any duplication or distribution of the information contained herein is strictly prohibited.

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